Ich sehe so viele Posts mittlerweile, bei denen ich mich frage ob das nicht KI generierter Content ist. Manchmal ist es offensichtlich, manchmal weisen einen andere Nutzer drauf hin.

Aber im großen Ganzen habe ich das Gefühl es wird immer mehr. Und den Kommentaren kann man auch teilweise nicht mehr trauen.

Das ist für mich ein Grund die Webseite nicht mehr zu nutzen. Warum soll ich mir Fake-Posts anschauen und drunter die Fake-Kommentare lesen.

Ich nutze Reddit wie viele als Unterhaltungs-; und Nachrichtenquelle. Aber ich fand es auch immer gut ein bisschen ein Gefühl für andere Meinungen bei aktuellen Themen zu bekommen. Reddit ist für mich noch einer der "gepflegteren" Social Media Seiten aber das scheint sich gerade irgendwie aufzulösen.

TL;DR: 1 Year after infection from almost severe to moderate bordering on mild. LDA 0,5 mg and LDN 1,5 mg + Magnesium help my Sensitivitys/ Fatigue and SFN.

M26, Vaccined multiple Infections causing weak immunesystem and food intolerances with another Covid-Infection causing Post Viral Fatigue in May 2025.

• PEM
• small fiber neuropathy
• POTS
• leaky gut syndrome
• weak immune system
• heat dysautonomia (extreme body temperature changes based on daytime)
• food intolerances
• extreme sensitivity to light/sound/touch/smell/human presence

- no histamine-intolerance
- no MCAS
- no brainfog
- minor insomnia

what i take

• low dose naltrexon (LDN) 1,5mg -> 0,5mg 1hour before lunch and 1mg before bedtime
• low dose aripiprazole (LDA) 1mg before bedtime
• prebiotics to restore the gut (probiotics gave to heavy sideeffects so I had to give them up)
• melatonin
• magnesium

After 1 year

• baseline up for PEM (from bordering on severe to comfortably moderate)
• small fiber neuropathy small improvement
• POTS unchanged
• leaky gut syndrome small improvement
• immune system unchanged
• heat dysautonomia major improvement
• food intolerance small improvement
• Light sensitivity way down (screentime from 10 min a day to >3 hours). Still in a dark room
• Touch sensitivity & human presence issues gone

Storytime:

Like many, I developed health issues from coid but didnt make the connection. In 2024 I got sick every 5 weeks even in summer. New food Intolerances etc.. While traveling I got reinfected and had multiple PEMs in a foreign Country... I was hospitalized once and went to the ER twice. After 3 Weeks isolated in a hotel I managed to fly back to germany and instantly felt way better from the better climate, air and the psychological relief to be home.

Then I pushed it to soon again because of family troubles. And i crashed HARD.
Went from mild being able to cycle to almost bedbound within 2 weeks of rolling PEM. I only stabilized by giving up everything i could at the time. I spend days not looking at my phone, not doing more than walking to the toilet once a day.

I am very privileged and lucky to have basically every family member, doctor and friend believe me. Also This sub gave me enough understanding about what was going on with me. The hospital gave me a positive covid test. I also had a antibody test and a mitochondrial function test 1 month later.

Still i worsened slowly. I tested gut health, deficiencies and food intolerances. Turns out Covid wrecked my gut flora and therefore my body became unable to properly absorb all nutrients from food. Also I got leaky gut syndrome causing food intolerances. So I am also taking Prebiotics. I dont feel a big difference but I will keep taking them.

I started improving fatigue-wise by pacing and time. I feel guilty because there is nothing else really. I believe I only have post viral fatigue not cfs. Still the sfn and sensitivitys stayed/stay. Here is where LDN and LDA made a huge difference for me in the last 2 months (taking both since 5 months).

I still am a looong way from being healthy again and I know a since reinfection will ruin my progress.

Still I want to share my story for those that just got this illness or those not sure about LDN / LDA.

Regarding LDN: I cant hanlde the 1,5 mg in one go so I split it up to 0,5 mg before lunch (1hour) and 1 mg before bedtime.

Long COVID remains difficult to diagnose and treat, particularly in Belgium. A recent report estimated that this hidden epidemic will impose an annual societal cost of tens of billions of euros on OECD member countries. In the absence of clear biomarkers, many practitioners still sometimes mistakenly attribute some of the symptoms to psychosomatic causes. Since 2022, the team led by Prof. Charles Nicaise (Molecular Physiology Research Unit - Namur Research Institute for Life Sciences (NARILIS) - UNamur), notably through the work of Margaux Mignolet, a FRIA researcher and PhD student, has been exploring the hypothesis of immune dysregulation occurring during acute infection and leading to the production of autoantibodies directed against components of the nervous system.

Thirteen patients whose symptoms were consistent with long neurological COVID and were confirmed by tests assessing their cognitive and pain-related complaints were included in this study. 

After collecting blood samples, the researchers isolated the patients’ immunoglobulin G (IgG) antibodies and studied their effects in a passive transfer mouse model at Professor Charles Nicaise’s LNR laboratory. The animals underwent a battery of behavioural tests assessing, in particular, pain sensitivity thresholds, as well as other cognitive, anxiety, or depressive disorders.

Several major discoveries have been made

IgG transfer and pain: After transfer of IgG from patients, mice develop painful hypersensitivity, specifically mechanical allodynia—meaning that a tactile stimulus that is usually painless becomes painful—as well as thermal hyperalgesia—meaning that an uncomfortable hot or cold stimulus becomes very painful.

Specificity of the effect: the transfer of these IgG to laboratory mice does not induce cognitive (e.g., memory), anxiety, or depressive disorders, suggesting distinct mechanisms depending on the symptoms.

Proof of causality: when the antibodies are destroyed before injection, or when serum from which IgG has been removed is injected, the painful effect disappears.

Target of autoantibodies: IgG binds to the spinal ganglia along the spinal column, structures containing sensory neurons that relay information, for example, between the skin and the brain. Autoantibodies recognize peripheral neurons involved in pain (nociception) and the perception of body position or deep sensation (proprioception).

Source: https://www.unamur.be/en/newsroom/long-covid-unamur-chu-ucl-namur-unravels-biological-mechanism-behind-the-pain