> While some holdouts still favor a slow virus or even bacterial etiology of prion diseases (Manuelidis, Sklaviadis and Manuelidis 1987; Broxmeyer 2004; Bastian et al. 2007; Manuelidis 2007), most scientists now accept the premise that normal, host-encoded PrPCcan misfold into a pathologic form (PrPSc) to cause prion disease in susceptible hosts.
Apparently their objection is that diseases are the result of genetic processes and misfolded proteins aren't encoded for by the host animal.
To me that's a bit like not believing in lead poisoning because metal isn't alive. It's kinda incredible that even in such an arcane area of the sciences there's still that one out of ten dentists railing against Big Toothpaste.
I was gonna post this in today i learned but then I'd have to read their replies. Not gonna play myself that way.